http://note.pearltrees.com/14006750/93f36b2542d99d072d0e93ba1a7765d0 if you live in an apartment or house or just would like to ensure your home is free of EMFs, there are a number of ways you can reduce exposure. One of the most effective is to limit your electronic device use. You can also turn to EMF block paint to stop EMF radiation from reaching your house. Another way to shield your house from EMF radiations is to put up an RF shielding canopy. This is a fabric of net that contains EMF shielding. It's used to prevent EMFs from entering a room. Another option is to get your home fitted with an enclosure that is conductive. These enclosures are known as Faraday cages.
Several studies have shown that the non-ionizing RF EMF can cause antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. Hop over to this website could be the reason for the long-term responses seen in certain patients suffering from advanced HCC. However, the mechanism behind AM EMF's impact on cancer patients is not yet clear.
The effects from AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were split into three groups. First, the group that was unaffected RF EMF. Second group members were exposed to RF EMF at frequencies similar to the one used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was compared to that of RCF. https://postheaven.net/toasthoe5/emf-blocking-and-emf-shielding indicated that cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind tumour-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is caused through CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have wider implications for the treatment and diagnosis of different cancers.
The tumours of those in the group that were unaffected to RF EMF, and were fed a standard mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells after they were between five and seven weeks old. The tumours were then euthanized in cases of excessive burden.
The tumours from the three groups also showed distinct growth curves. The tumors treated with HCCMF saw a significant reduction in size of the tumor after eight weeks. However, the tumors treated with RCF did not show any signs of shrinkage. The difference was substantial. The tumors treated with RCF showed necrosis that is common in tumours that are exposed to RCF. There is a possibility that the necrosis was due to the lack of oxygen in larger cancers.
In conclusion, the findings indicate an AM-RF EMF exhibits anticancer properties in vitro as well as in vivo. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF produces these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting influence on the development of HCC tumours in vivo.